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C-superoxide dismutase Chain A, Crystal Structure Of Class I Chitinase GSH-dependent > 공지사항
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C-superoxide dismutase Chain A, Crystal Structure Of Class I Chitinase…

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작성자 Genia Haralson 작성일23-09-08 12:01 조회18회 댓글0건

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C-superoxide dismutase Chain A, Crystal Structure Of Class I Chitinase GSH-dependent dehydroascorbate reductase 1 Putative superoxide dismutase [Cu-Zn] L-ascorbate peroxidase 1, Triosephosphate isomerase Salt stress-induced protein Pathogenesis-related protein Bet vI Acidic PR-1 type pathogenesisrelated protein PR-1 Hypothetical protein Ascorbate peroxidase Enolase Enolase Hypothetical protein Hypothetical protein Hypothetical protein Chitinase Cytosolic malate dehydrogenase Salt stress-induced protein Hypothetical proteinAccession # gi|115452789 gi|115452789 gi|407472 gi|22296339 pdb|2DKV|A gi|6939839 gi|42408425 P93404 gi|553107 gi|158513205 gi|9230755 gi|12005673 gi|115461070 NP_001060741 gi|780372 Q42971 gi|115465323 gi|125557770 gi|125532459| gi|561873 gi|115482534 gi|158513205 EAYScore Mr (gel) pI (gel) Mr (cal) pI (cal) 138 65 56 26 30 43 72 99 49 88 55 182 87 74 88 74 98 37 73 51 66 104 38.0 39.0 36.0 15.0 37.0 26.0 17.0 28.0 28.0 11.0 18.0 17.5 29.0 32.0 60.0 50.0 60.0 45.0 25.0 36.5 37.5 39 6,7 6.6 6.6 6.4 6.2 6.1 5.5 5.5 5.4 5.0 4.9 4.2 4.7 5.2 5.4 5.4 5.2 5.7 4.2 4.2 5.9 5.3 39 39 35.5 14.7 32.6 23.5 20.5 27 27.5 15.1 16.6 17.5 27.2 27 47.9 47.9 58.8 95.7 24.5 34.3 35.5 15.1 40.6 6.3 6.3 7.3 5.9 5.8 5.6 5.7 5.4 6.6 5.1 4.9 4.5 5.0 5.2 5.4 5.4 5.9 8.0 4.6 4.4 5.75 5.1 8.WAPSPADAAAGR EHGAPQDENR GPIQLSFNFNYGPAGR AAVGHPDTLGDCPFSQR GTSQVEGVVTLTQDDQGP TTVNVR VATPDQAQEVHDGLR EFSIPLQDSGHVVGFFGR MIEDYLVAHPAEYA ADVGVGPVSWDDTVAAY AESYAAQR WWDTFPANVDGAR MTAEIGEQVQIVGDDLLVT NPTRKADATVAGDDR AGYAPPHWVQPGQGDR ELFEQLLLHR ELVADDEWLNTEFISTVQQ R EFSIPLQDSGHVVGFFGR
Hafez et al. World Journal of Surgical Oncology 2010, 8:76 http://www.wjso.com/content/8/1/WORLD JOURNAL OF SURGICAL ONCOLOGYREVIEWOpen AccessThe safety and efficacy of gamma knife surgery in management of glomus jugulare tumorRaef FA Hafez*, Magad S Morgan, Osama M FahmyAbstractBackground: Glomus jugulare is a slowly growing, locally destructive tumor located in the skull base with difficult surgical access. The operative approach is, complicated by the fact that lesions may be both intra and extradural with engulfment of critical neurovascular structures. The tumor is frequently highly vascular, thus tumor resection entails a great deal of morbidity and not infrequent mortality. At timeslarge residual tumors are left behind. To decrease the morbidity associated with surgical resection of glomus jugulare, gamma knife surgery (GKS) was performed as an alternative in 13 patients to evaluate its safety and efficacy. Methods: A retrospective review of 13 residual Alectinib or unresectable glomus jagulare treated with GKS between 2004 and 2008. Of PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/16989806 these, 11 patients underwent GKS as the primary management and one case each was treated for postoperative residual disease and postembolization. The radiosurgical dose to the tumor margin ranged between 12-15 Gy. Results: Post- gamma knife surgery and during the follow-up period twelve patients demonstrated neurological stability while clinical improvement was achieved in 5 patients. One case developed transient partial 7th nerve palsy that responded to medical treatment. In all patients radiographic MRI follow-up was obtained, the tumor size decreased in two cases and remained stable (local tumor control) in eleven patients. Conclusions: Gamma knife surgery provids tumor control with a lowering of risk of developing a new cranial nerve injury in early follow-up period. This procedure can be safely used as a primary management tool in patients with glomus.

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